Author

Cecilia Lundberg

Lund UniversityORCID 0000-0002-6959-2564Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:benchmarking· 1 Research Focus:cell type annotation· 1 Assay:CRISPR screen· 1 Research Focus:differential expression· 1 Assay:electronic health records· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

Luís QuintinoiD· 3 shared Elin ArvidssoniD· 2 shared Ermelinda Sabarese· 2 shared Alexander SvanbergssoniD· 1 shared Diana Duarte LoboiD· 1 shared Elsa Pi Börjesson· 1 shared Fábio Teixeira DuarteiD· 1 shared Gunnar K. GourasiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 23 mutual co-author links

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3 shared papersMolecular Biology

Luís QuintinoORCID

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3 papers

Recent·Most cited

CRISPRa-mediated activation of Tfeb in a fibril-induced alpha-synuclein aggregation model

Elin Arvidsson, Elsa Pi Börjesson, Ermelinda Sabarese, Luís Quintino, Cecilia Lundberg

The paper uses CRISPRa to activate Tfeb in a rat model of alpha-synuclein aggregation but finds limited protein expression and no additive effects with media treatments.

bioRxiv · Oct 19, 2026 · PreprintMolecular Biology

A systematic screening assay identifies efficient small guide RNAs for CRISPR activation

Elin Arvidsson, Diana Duarte Lobo, Ermelinda Sabarese, Fábio Teixeira Duarte, Rui Jorge Nobre, Cecilia Lundberg, Luís Quintino

The paper presents a screening assay to identify efficient sgRNAs for CRISPR activation and provides a cloning workflow for viral vector integration.

Frontiers in Bioengineering and Biotechnology · Jan 23, 2025Peer-reviewedMolecular Biology

Aβ/APP-induced hyperexcitability and dysregulation of homeostatic synaptic plasticity in models of Alzheimer's disease

Isak Martinsson, Luís Quintino, Megg G. Garcia, Sabine C Konings, Laura Torres-Garcia, Alexander Svanbergsson, Oliver Stange, Rebecca England, Tomas Deierborg, Jiayi Li, Cecilia Lundberg, Gunnar K. Gouras

This study shows that APP and Aβ increase neuronal hyperexcitability and impair homeostatic synaptic and intrinsic plasticity in Alzheimer's disease models.

Frontiers in Aging Neuroscience · Jul 6, 2022Peer-reviewedNeuroscience