Author

Nia G. Hammond

University of ChicagoORCID 0000-0002-7120-0486Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:statistical model· 1

Frequent Co-Authors

Alpaslan TasdoganiD· 1 shared Brandon FaubertiD· 1 shared Brianna K. ChangiD· 1 shared Christine M. BestvinaiD· 1 shared Elyse G. SchechteriD· 1 shared Hardik ShahiD· 1 shared Juliya HsiangiD· 1 shared Kristen Erika ShemaiD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCancer Biology

Alpaslan TasdoganORCID

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1 paper

Recent·Most cited

MCT1 activity defines an aggressive metabolic phenotype and therapeutic target in non-small cell lung cancer

Robert B Cameron, Kristen Erika Shema, Nathan Dubois, Maia G. Clare, Nia G. Hammond, Mayher Kaur, Brianna K. Chang, Elyse G. Schechter, Juliya Hsiang, Zuzanna Marczenia, Marina C. Garassino, Ling Cai, Christine M. Bestvina, Thomas P. Mathews, Alpaslan Tasdogan, , Brandon Faubert

This study shows that MCT1-mediated lactate uptake sustains nucleotide homeostasis in NSCLC, and its inhibition enhances chemotherapy efficacy by exposing a metabolic vulnerability.

bioRxiv · Sep 22, 2026 · PreprintCancer Biology