Author

Carlotta Bon

Italian Institute of TechnologyORCID 0000-0002-7443-4659Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Assay:long-read sequencing· 1 Organism:mouse· 1

Frequent Co-Authors

Stefano GustincichiD· 2 shared Abraham Tettey‐MateyiD· 1 shared Andrea ArmirottiiD· 1 shared Bianca PierattiniiD· 1 shared Clarissa BracciaiD· 1 shared Claudio SantoroiD· 1 shared Codrich Marta· 1 shared Devid DamianiiD· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersMolecular Biology

Stefano GustincichORCID

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2 papers

Recent·Most cited

Internal Ribosome Entry Sites act as Effector Domain in linear and circular antisense long non-coding SINEUP RNAs

Sabrina D’Agostino, Abraham Tettey‐Matey, Massimiliano Volpe, Bianca Pierattini, Federico Ansaloni, Pierre Lau, Carlotta Bon, Omar Peruzzo, Clarissa Braccia, Andrea Armirotti, Margherita Scarpato, Devid Damiani, Valerio Di Carlo, Laura Broglia, Elías Bechara, , Stefano Gustincich

This study identifies SINEUP RNAs with Internal Ribosomal Entry Site (IRES) sequences as effector domains, enhancing translation of target mRNAs through both linear and circular transcripts.

bioRxiv · May 25, 2023 · PreprintMolecular Biology

Neuronal hemoglobin induces loss of dopaminergic neurons in mouse Substantia nigra, cognitive deficits and cleavage of endogenous α-synuclein.

Santulli Chiara, Carlotta Bon, Elena De Cecco, Codrich Marta, Joanna Narkiewicz, Parisse Pietro, Fabio Perissinotto, Santoro Claudio, Persichetti Francesca, Legname Giuseppe, Espinoza Stefano, Stefano Gustincich

This study shows that neuronal hemoglobin overexpression in mice leads to dopaminergic neuron loss, cognitive deficits, and cleavage of α-synuclein.

bioRxiv · Oct 13, 2021 · PreprintNeuroscience