Author

Yoshitaka Hayashi

Research Institute of Environmental Medicine, Nagoya UniversityORCID 0000-0002-7557-4303Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Assay:proteomics· 1

Frequent Co-Authors

Atsushi SuzukiiD· 1 shared Ayako FukunakaiD· 1 shared Eisuke Tomatsu· 1 shared Izuho HatadaiD· 1 shared Masaki KobayashiiD· 1 shared Mutsumi Habara· 1 shared Naoya MuraoiD· 1 shared Norihide YokoiiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperPhysiology

Atsushi SuzukiORCID

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2 papers

Recent·Most cited

PhysiologybioRxivPreprintApr 1, 2025

Intestinal fructose metabolism triggers a GLP-1-β-cell axis to prevent post-fructose hyperglycaemia

Naoya Murao, Yusuke Seino, Risa Morikawa…·Atsushi Suzuki

This study shows that intestinal fructose metabolism increases GLP-1 secretion, which enhances insulin release and prevents hyperglycaemia, with this effect being impaired in obese diabetic mice.

DOI 10.1101/2025.04.01.646509

mouseproteomics

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Cell BiologybioRxivPreprintFeb 27, 2025

Glucagon dysfunction in the liver induces hyperplasia of PP cells and the production of glucagon and pancreatic polypeptide double-positive cells

Yuko Nakagawa, Takuro Horii, Ayako Fukunaka…·Yoshio Fujitani

The paper shows that glucagon dysfunction in the liver leads to PP cell hyperplasia and the emergence of GCG+ PP+ double-positive cells through hyperaminoacidemia and mTOR signaling.

DOI 10.1101/2025.02.27.640454

mouseexperimental assay

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