Author

Frank Zaucke

Goethe University FrankfurtORCID 0000-0002-7680-9354Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 2 Research Focus:differential expression· 1 Approach:experimental assay· 1 Assay:proteomics· 1 Approach:statistical model· 1 Organism:zebrafish· 1

Frequent Co-Authors

Adriana Figueroa GarciaiD· 1 shared Chantal FayadiD· 1 shared Daniela WelckeriD· 1 shared Dimitrije MilunoviD· 1 shared Douglas R. KeeneiD· 1 shared Gerhard SengleiD· 1 shared Giulio GattoiD· 1 shared Hans‐Martin PogodaiD· 1 shared

Activity by Year

2022-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperGenetics

Adriana Figueroa GarciaORCID

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2 papers

Recent·Most cited

Mitochondrial dysfunction plays a key role in premature musculoskeletal aging in hypochondroplasia mouse model

Chantal Fayad, Giulio Gatto, Stéphane Blouin, Michal Tamáš, Markus A. Hartmann, Dimitrije Milunov, Ida Chiara Guerrera, Soham Saha, Adriana Figueroa Garcia, Paolo Bonaldo, Frank Zaucke, Nadja Fratzl‐Zelman, Laurence Legeai‐Mallet

This study shows that mitochondrial dysfunction contributes to premature musculoskeletal aging in HCH mice, with proteomics revealing altered aging trajectories and bone fragility.

bioRxiv · Oct 22, 2026 · PreprintGenetics

Vertebrate extracellular matrix protein hemicentin-1 interacts physically and genetically with basement membrane protein nidogen-2

Jin-Li Zhang, Stefânia Konrad Richetti, Thomas Ramezani, Daniela Welcker, Steffen Lütke, Hans‐Martin Pogoda, Julia Hatzold, Frank Zaucke, Douglas R. Keene, Wilhelm Bloch, Gerhard Sengle, Matthias Hammerschmidt

This study identifies nidogen-2 as a physical and genetic interaction partner of hemicentin-1, showing their cooperative role in basement membrane integrity.

Matrix Biology · Sep 1, 2022Peer-reviewedDevelopmental Biology