Author

Christopher D. Wiley

Tufts UniversityORCID 0000-0002-7699-6866Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1 Research Focus:metabolism & bioenergetics· 1

Frequent Co-Authors

A. H. Kung· 1 shared Aleksandra Aljakna Kahn· 1 shared Amene MajidipuriD· 1 shared Andrew S. GreenbergiD· 1 shared Arpan G. MazumderiD· 1 shared Arvind Ramanthan· 1 shared Bronwyn A. Mogck· 1 shared Carlyn Guthrie· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

A. H. Kung

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Author profile →

2 papers

Recent·Most cited

Fatty acid desaturation guides cellular decisions between ferroptosis and cellular senescence

Chisaka Kuehnemann, Samantha T. Jezak, Bronwyn A. Mogck, Niharika Kura, Rushmeen Tariq, Yuheng Che, Maija Vaittinen, Ioannis Siokas, Sharvari Deshpande, Carlyn Guthrie, Xuejuan Tan, Eliézer Aïmontché, Aleksandra Aljakna Kahn, Peter Reifsnyder, Gung Lee, , Christopher D. Wiley

The paper shows that fatty acid desaturation, specifically DGLA, influences cellular decisions between ferroptosis and senescence, with implications for aging and obesity.

Cell · Nov 1, 2026Peer-reviewedCell Biology

Targeting Mitochondrial Dysfunction with Mdivi-1 Confers Therapeutic Protection in a Mouse Model of Mustard Keratopathy

Arpan G. Mazumder, Evan Magarychoff, Hamid Alemi, Rahul Raghav, Michael T. Chin, Zhen Yan, Christopher D. Wiley, M. Elizabeth Fini

This study identifies mitochondrial dysfunction as a key mechanism in mustard keratopathy and shows that inhibiting DNM1L with Mdivi-1 protects against corneal injury in mice.

bioRxiv · Sep 2, 2026 · PreprintMolecular Biology