Author

Thomas Mortimer

Institute for Research in BiomedicineORCID 0000-0002-7822-4093Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1

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Research Focus:cell type annotation· 1 Assay:ChIP-seq· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Camille Stephan‐Otto AttoliniiD· 1 shared Guiomar SolanasiD· 1 shared Júlia BonjochiD· 1 shared Laura Álvarez· 1 shared Óscar ReinaiD· 1 shared Paloma SoláiD· 1 shared Salvador Aznar BenitahiD· 1 shared Sara A. WickströmiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperMolecular Biology

Camille Stephan‐Otto AttoliniORCID

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1 paper

Recent·Most cited

BMAL1 and YAP cooperate to hijack enhancers and promote inflammation in the aged epidermis

Júlia Bonjoch, Paloma Solá, Óscar Reina, Thomas Mortimer, Yekaterina A. Miroshnikova, Sara A. Wickström, Camille Stephan‐Otto Attolini, Laura Álvarez, Salvador Aznar Benitah, Guiomar Solanas

This study identifies a BMAL1-YAP transcriptional complex that hijacks enhancers in aged epidermis to promote inflammation by rewiring chromatin dynamics and co-regulating inflammation-related genes with NF-KB.

bioRxiv · Apr 22, 2025 · PreprintMolecular Biology