Author

Mikhail Rudinskiy

ETH ZurichORCID 0000-0002-7915-5616Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Approach:biophysical modeling· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Assay:proteomics· 1

Frequent Co-Authors

Maurizio MolinariiD· 2 shared Aida DelgadoiD· 1 shared Ana María García-CollazoiD· 1 shared Ana Ruano· 1 shared Andrea RaimondiiD· 1 shared Carmela Galli· 1 shared Elena CuberoiD· 1 shared Ilaria FregnoiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersCell Biology

Maurizio MolinariORCID

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2 papers

Recent·Most cited

Cell BiologyNature Cell BiologyPeer-reviewedAug 4, 2025

ER-phagy Receptors Intrinsically Disordered Modules Drive ER Fragmentation and ER-phagy

Mikhail Rudinskiy, Carmela Galli, Andrea Raimondi, Maurizio Molinari

This study shows that intrinsically disordered regions (IDRs) of ER-phagy receptors drive ER fragmentation, not their membrane-remodeling domains.

16 citationstop 3%

proteomicsexperimental assay

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Cell BiologyInternational Journal of Molecular SciencesPeer-reviewedMay 6, 2025

A novel class of allosteric glucosylceramidase beta 1 correctors that reduce cellular stress and enhance lysosomal function

Ilaria Fregno, Natàlia Pérez‐Carmona, Mikhail Rudinskiy…·Maurizio Molinari

This paper develops two allosteric GCase correctors that improve folding and lysosomal function in GD patient fibroblasts, reducing cellular stress and offering potential therapy for GD, PD, and Dementia with Lewy Bodies.

10 citationstop 3%

humanexperimental assay

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