Author

Meng Wang

Army Medical UniversityORCID 0000-0002-8168-8999Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Meng Wang13 papers · Chinese Academy of Sciences

Biology Disciplines

Click to filter author papers

Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Shaofan HuiD· 2 shared Yiguo ZhangiD· 2 shared Da Lyu· 1 shared Feng Jing· 1 shared Liu Keli· 1 shared Lu QiuiD· 1 shared Reziyamu WufueriD· 1 shared Rongzhen Deng· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

8 top collaborators · 19 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Shaofan HuORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJul 9, 2025

Curcumin activates distinct programming of redox metabolism towards differential regulation of gene expression mediated by Nrf1 and Nrf2

Reziyamu Wufuer, Feng Jing, Shaofan Hu…·Yiguo Zhang

This study investigates how curcumin modulates Nrf1 and Nrf2 expression and their roles in redox metabolism and gene regulation in HepG2 cells and xenograft tumors.

DOI 10.1101/2025.07.09.663854

humanbulk RNA-seq

𝕏 PostBluesky

BiochemistryBiochimica et Biophysica Acta (BBA) - Molecular Cell ResearchPeer-reviewedApr 23, 2025

Deletion of Nrf1 exacerbates oxidative stress-induced cellular senescence by disrupting the cell homeostasis

Da Lyu, Meng Wang, Lu Qiu…·Yiguo Zhang

This study shows that deleting Nrf1 increases oxidative stress-induced cellular senescence by disrupting homeostasis, as evidenced by increased senescence markers and impaired autophagy.

5 citations

mouseproteomics

𝕏 PostBluesky