Author

Ming Li

Kunming Institute of ZoologyORCID 0000-0002-8197-6552Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Ming Li14 papers · McGovern Institute for Brain Research

Biology Disciplines

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Genetic & Genomic Medicine· 1 Genetics· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:GWAS / association mapping· 2 Assay:GWAS summary statistics· 2 Organism:human· 2 Approach:statistical model· 2 Research Focus:eQTL / molecular QTL mapping· 1 Approach:experimental assay· 1 Research Focus:fine-mapping· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Xiong‐Jian LuoiD· 2 shared Yong‐Gang YaoiD· 2 shared Aihui Tang· 1 shared Brittany L. MitchelliD· 1 shared C WangiD· 1 shared Chang XuiD· 1 shared Chunhua Zeng· 1 shared Chunli SuniD· 1 shared

Activity by Year

2021-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 18 mutual co-author links

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2 shared papersGenetic & Genomic Medicine

Xiong‐Jian LuoORCID

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Author profile →

3 papers

Recent·Most cited

(2R,6R)-Hydroxynorketamine elicits rapid antidepressant effects by promoting astrocytic μ-δ opioid receptor heterodimerization

Yanxia Liang, Lingjun Wang, Yajie Li, Xiaoxue Li, Y Sun, Mengli Yang, Xuzhuo Guo, Junxu Mu, Chang Xu, Rupak Thapa, Ye Cheng, Huiqiang Zhang, Zecong He, Shi Yan, S M Yang, , Xiang Cai

This study identifies HNK's mechanism of action through astrocytic μ-δ opioid receptor heterodimerization, showing it restores these heterodimers and produces rapid antidepressant effects.

bioRxiv · Jun 3, 2026 · PreprintNeuroscience

Cross-ancestry genome-wide association study and systems-level integrative analyses implicate new risk genes and therapeutic targets for depression

Yifan Li, Xinglun Dang, Rui Chen, Zhaowei Teng, Junyang Wang, S. L. Li, Yingying Yue, Brittany L. Mitchell, Yong Zeng, Yong‐Gang Yao, Ming Li, Zhongchun Liu, Yonggui Yuan, Tao Li, Zhijun Zhang, Xiong‐Jian Luo

This study identifies 287 depression risk loci, 140 new, through cross-ancestry GWAS and integrates functional and pathway analyses to highlight potential therapeutic targets.

Nature Human Behaviour · Feb 24, 2025Peer-reviewedGenetic & Genomic Medicine

A multiple-causal-gene-cluster model underlying GWAS signals of Alzheimer's disease

Min Xu, Qianjin Liu, Rui Bi, Yu Li, Chunhua Zeng, Zhongjiang Yan, Quanzhen Zheng, Xiao Li, Chunli Sun, Maosen Ye, Xiong‐Jian Luo, Ming Li, Dengfeng Zhang, Yong‐Gang Yao

The paper identifies multiple causal genes and variants within GWAS loci for Alzheimer's disease and proposes a model where co-dysregulation of gene clusters contributes to disease development.

bioRxiv · May 14, 2021 · PreprintGenetics