Chloe M. Hall

This study shows that GAD67-GFP mice with GABA deficiency develop thermal allodynia after nerve injury, while wildtype mice do not, indicating GABA's role in neuropathic pain.

This study shows that CLN3 is essential for synaptic function in Batten disease and that early gene therapy can reverse synaptic deficits.

This study investigates the effect of sodium channel inhibitors on plaque and tangle development in Alzheimer's mouse models, finding reduced tangle density with inhibitor treatment.