Kaci T. Mitchum

This study investigates how metabolic flexibility and energy substrate utilization affect myometrial contractility in human cells, finding a shift from oxidative phosphorylation to glycolysis during oxytocin-induced contractions.

This paper introduces EMMIs, engineered myometrial microtissues, to directly measure oxytocin-induced contractility and show that prolonged oxytocin exposure reduces contractile force.

This study profiles S1P metabolic enzymes and receptors in human gestational tissues across pregnancy, finding tissue-specific expression and abundance patterns.