Author

Saul S. Siller

Yale UniversityORCID 0000-0002-8299-0794Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:metabolomics· 1 Assay:metagenomics· 1

Frequent Co-Authors

Jessica NouwsiD· 1 shared John E. McDonoughiD· 1 shared Juan IovannaiD· 1 shared Maor SauleriD· 1 shared Patricia Santofimia‐CastañoiD· 1 shared Sang‐Hun KimiD· 1 shared Sojin KimiD· 1 shared Tao YangiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperCell Biology

Jessica NouwsORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

The ferroptosis inhibitor NUPR1 coordinates the mitochondrial response to oxidative stress and cell metabolism during COPD pathogenesis in the lung

Saul S. Siller, Tao Yang, Jessica Nouws, Sojin Kim, Sang‐Hun Kim, Patricia Santofimia‐Castaño, Juan Iovanna, John E. McDonough, Maor Sauler

This study shows NUPR1 inhibition worsens oxidative stress, mitochondrial dysfunction, and metabolic imbalance in COPD, linking it to ferroptosis and disease progression.

bioRxiv · Apr 27, 2025 · PreprintCell Biology