Author

Hagay Akiva

Weizmann Institute of ScienceORCID 0000-0002-8333-6209Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Organism:mouse· 3 Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Research Focus:differential expression· 1 Organism:human· 1 Assay:proteomics· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Valery KrizhanovskyiD· 3 shared Noa RachmianiD· 2 shared Tommaso CroeseiD· 2 shared Ulysse CherquiiD· 2 shared Alexander KertseriD· 1 shared Angham Ibraheem· 1 shared Avi MayoiD· 1 shared Bar Nathansohn· 1 shared

Activity by Year

2023-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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3 shared papersNeuroscience

Valery KrizhanovskyORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

NeurosciencebioRxivPreprintDec 24, 2025

Systemic administration of PD-L1 blocking antibodies leads to removal of senescent microglia

Yuliya Androsova, Alexander Kertser, Hannah Partney…·Michal Schwartz

The paper shows that systemic PD-L1 blockade removes senescent microglia in mice, reducing neuroinflammation and suggesting a therapeutic role in Alzheimer's disease.

1 citation

mouseexperimental assay

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Cell BiologybioRxivPreprintNov 14, 2025

Single-cell quantification of senescence burden reveals cell type-specific ageing dynamics across organs

Ulysse Cherqui, Ilanit Sopher, Hagay Akiva…·Valery Krizhanovsky

This study quantifies senescence burden at the single-cell level across tissues in mice and human PBMCs, revealing tissue-specific ageing dynamics and senescence marker heterogeneity.

1 citation

mousehuman

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NeurosciencebioRxivPreprintMar 20, 2023

TREM2-dependent senescent microglia conserved in aging and Alzheimer's disease

Noa Rachmian, Silvia Medina, Ulysse Cherqui…·Michal Schwartz

The paper identifies TREM2-dependent senescent microglia in aging and Alzheimer's disease, showing that TREM2 inhibition reduces their abundance and improves cognitive deficits.

6 citations

mouseproteomics

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