Mayu Inaba

The paper shows that transposable elements in Y-linked fertility genes of Drosophila are transcribed as part of nascent transcripts and contribute to Y-loop structure and male fertility.

The paper shows how BMP signaling produces opposing effects on stem cells and differentiating cells through regulation of Brinker, maintaining tissue homeostasis in Drosophila germline.

This study uses high-resolution imaging to examine chromosome territory organization during zygotic genome activation, revealing large-scale homolog pairing and transcriptional dynamics in single nuclei.

The paper identifies dedifferentiated stem cells in Drosophila testis using a new genetic tool and live imaging, showing lineage-specific promoters are critical for detection.

The paper examines how asymmetric stem cell division affects clonal dynamics in Drosophila germline stem cells, finding that it maintains genetic heterogeneity and prevents clonal expansion.

The paper shows that BMP ligand diffuses from the niche and has opposing roles in maintaining stem cells and promoting differentiation of their daughter cells.

This study identifies BMP signaling from somatic cells to germ cells as essential for proper spermiogenesis in Drosophila, with defects in sperm head bundling and mitochondrial morphology observed upon disruption.

The paper shows dedifferentiating germ cells in Drosophila re-enter mitosis with correct spindle orientation and activate a centrosome orientation checkpoint to ensure asymmetric division.

The paper shows that changes in homologous chromosome pairing at the Stat92E locus are required for transcriptional downregulation during Drosophila stem cell differentiation.

This study shows that microtubule acetylation is required for maintaining the barrier between adjacent tissues during oogenesis in Drosophila.

The paper shows that Mad dephosphorylation at the nuclear envelope creates asymmetry in pMad levels between stem cells and differentiating daughter cells, preventing tumor formation.

The paper shows that MT-nanotubes in Drosophila GSCs restrict niche signaling range by enabling specific receptor-ligand interaction and subsequent degradation of the signaling components.