Yu-Chiao Lin

This paper investigates the role of KCNT1 mutations in Ca2+ homeostasis and cardiomyocyte function, showing that its cytoplasmic tail modulates ER-PM contacts and Ca2+ dynamics independently of its ion-conducting function.

This study shows that mitochondria-ER contacts inhibit store-operated Ca²⁺ entry by trapping STIM1 through Ca²⁺ flickering and microdomain formation.

This study shows SLK and Ezrin affect actin cytoskeleton organization independently of phosphorylation, revealing non-enzymatic roles for Ezrin in cytoskeletal dynamics.