Lisa Anne Taneyhill

The paper uses proteomic profiling to identify molecular changes in Elp1-deficient trigeminal ganglia, revealing disruptions in neurotrophic and metabolic pathways in a familial dysautonomia mouse model.

The paper identifies multiple glycoforms of TrkA and their interaction with N-cadherin during trigeminal ganglion development, suggesting a role in neurotrophic signaling and cell adhesion.

This study shows that N-cadherin is essential for trigeminal ganglion development, axon outgrowth, and target innervation by placodal and neural crest-derived neurons.

This study uses RNA-sequencing to analyze chick trigeminal ganglion development, identifying key pathways in gangliogenesis and neuron maturation.

The paper investigates Elp1's role in trigeminal ganglion development by knocking down Elp1 in chick placode cells, revealing its critical function in ganglion size and neuronal interactions.

This study shows that Neurog2 and NeuroD1 are essential for proper development of the chick trigeminal ganglion and its nerve branches.

This study shows that loss of Elp1 in trigeminal ganglion neurons leads to impaired axon outgrowth, reduced target innervation, and decreased TrkA neuron survival, explaining facial pain and temperature sensation loss in Familial Dysautonomia.

This study shows that Cx43 is involved in maintaining the size of the chick premigratory cranial neural crest cell population before EMT, without affecting EMT itself.

This study identifies Cadherin-7's role in neural crest cell-placodal neuron interactions during trigeminal ganglia formation in chick embryos.