Author

Han-Ying Huang

Sun Yat-sen UniversityORCID 0000-0002-8635-4867Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Chuang ZhaoiD· 2 shared Lin Hai TianiD· 2 shared Xin-Nan ZhengiD· 2 shared Huai‐Qiang JuiD· 1 shared Jia‐Xing YueiD· 1 shared Kai YuiD· 1 shared Shi Yan-xia· 1 shared Yan-Zhou CheniD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 31 mutual co-author links

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2 shared papersCancer Biology

Chuang ZhaoORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Targeting the sinusoidal-to-capillarized transition to normalize vessels in breast cancer liver metastasis

Han-Ying Huang, Chuang Zhao, Xin-Nan Zheng, Shi Yan-xia, Lin Hai Tian

This study identifies Esm1 as a target for normalizing dysfunctional vessels in liver metastasis by showing its role in capillary tip cells and its correlation with poor prognosis.

bioRxiv · Oct 29, 2026 · PreprintCancer Biology

Availability of an inflammatory macrophage niche drives phenotypic and functional alterations in Kupffer cells

Han-Ying Huang, Yan-Zhou Chen, Chuang Zhao, Xin-Nan Zheng, Kai Yu, Jia‐Xing Yue, Huai‐Qiang Ju, Yanxia Shi, Lin Hai Tian

The paper shows that blocking monocytes alone is insufficient to reduce LMAMs, as KCs can replenish them through proliferation or infiltration, and their reprogramming contributes to immunosuppression.

Nature Communications · Oct 29, 2024Peer-reviewedImmunology