Author

Catalina Matias

Indiana University IndianapolisORCID 0000-0002-8751-6530Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:proteomics· 1

Frequent Co-Authors

Jeffrey J. BraultiD· 2 shared Alan H. BeggsiD· 1 shared Behzad MoghadaszadehiD· 1 shared Elizabeth A. Sierra PotchanantiD· 1 shared Emily T Hickey· 1 shared Jeffrey J. WidrickiD· 1 shared Joshua R. HuotiD· 1 shared Kathryn M. YammineiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersGenetics

Jeffrey J. BraultORCID

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2 papers

Recent·Most cited

GeneticsHuman Molecular GeneticsPeer-reviewedNov 20, 2025

Loss of adenylosuccinate synthetase 1 in mice recapitulates features of ADSS1 myopathy

M. Kim, Kathryn M. Yammine, Emily T Hickey…·Alan H. Beggs

This paper presents a mouse model of ADSS1 myopathy by knocking out Adss1 and characterizes its motor deficits and histopathological features.

2 citations

mouseexperimental assay

𝕏 PostBluesky

Cell BiologybioRxivPreprintOct 31, 2025

Deficient Cardiolipin Remodeling Alters Muscle Fiber Composition and Neuromuscular Connectivity in Barth Syndrome

Catalina Matias, Paige L. Snider, Elizabeth A. Sierra Potchanant…·Jeffrey J. Brault

This study shows that Tafazzin deficiency in skeletal muscle causes neuromuscular remodeling, including fiber type shifts and motor unit loss, without resting energetic failure.

DOI 10.1101/2025.10.31.685856

mouseproteomics

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