Author

Elizabeth A. Sierra Potchanant

Indiana University School of MedicineORCID 0000-0003-0040-5916Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Catalina MatiasiD· 1 shared Jeffrey J. BraultiD· 1 shared Jesse B. TrejoiD· 1 shared Joshua R. HuotiD· 1 shared Marina GritsenkoiD· 1 shared Mark R. KelleyiD· 1 shared Melissa L. FisheliD· 1 shared Michael T. ChiniD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperCell Biology

Catalina MatiasORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintAug 19, 2026

Integrated Post-Translational Modification (PTM) Proteomics Reveals Early Redox and Mitochondrial Remodeling Following Ref-1 Inhibition in Pancreatic Cancer

Silpa Gampala, xiaolu Li, Jesse B. Trejo…·Mark R. Kelley

This paper uses integrated PTM proteomics to show that Ref-1 inhibition rapidly remodels redox and mitochondrial signaling in pancreatic cancer cells.

DOI 10.64898/2026.08.15.745014

humanproteomics

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Cell BiologybioRxivPreprintOct 31, 2025

Deficient Cardiolipin Remodeling Alters Muscle Fiber Composition and Neuromuscular Connectivity in Barth Syndrome

Catalina Matias, Paige L. Snider, Elizabeth A. Sierra Potchanant…·Jeffrey J. Brault

This study shows that Tafazzin deficiency in skeletal muscle causes neuromuscular remodeling, including fiber type shifts and motor unit loss, without resting energetic failure.

DOI 10.1101/2025.10.31.685856

mouseproteomics

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