Author

Samuel G. Brill-Weil

National Institutes of HealthORCID 0000-0002-9018-7109Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:electronic health records· 1 Organism:human· 1 Assay:imaging· 1 Organism:non-human primate· 1

Frequent Co-Authors

Paul F. KrameriD· 2 shared Renshu Zhang· 2 shared Zayd M. KhaliqiD· 2 shared Alex C. Cummins· 1 shared Amy Hauck NewmaniD· 1 shared Anna M. LipkiniD· 1 shared Anthony YaneziD· 1 shared Bruno B. AverbeckiD· 1 shared

Activity by Year

2022-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersNeuroscience

Paul F. KramerORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Presynaptic GABAA receptors control integration of nicotinic input onto dopaminergic axons in the striatum

Samuel G. Brill-Weil, Paul F. Kramer, Anthony Yanez, Anna M. Lipkin, Faye H. Clever, Renshu Zhang, Zayd M. Khaliq

The paper investigates how GABAARs on dopaminergic axons modulate nicotinic input integration, showing that GABAAR antagonism enhances nAChR-evoked activity.

Cell Reports · Nov 27, 2025Peer-reviewedNeuroscience

Fast-synaptic axo-axonal transmission from striatal cholinergic interneurons onto dopamine fibers.

Paul F. Kramer, Samuel G. Brill-Weil, Alex C. Cummins, Renshu Zhang, Gisela Andrea Camacho‐Hernandez, Amy Hauck Newman, Mark A. G. Eldridge, Bruno B. Averbeck, Zayd M. Khaliq

The paper demonstrates fast synaptic-like transmission from striatal cholinergic interneurons to dopaminergic axons, showing axonal integration of synaptic input into spiking.

Neuron · Aug 4, 2022Peer-reviewedNeuroscience