Anthony Yanez

The paper identifies dopamine as a direct agonist of GluDs, showing G-protein regulation of their activity and implications for neurological diseases.

This study shows that nicotinic receptors modulate dopamine signaling by both enhancing and suppressing axonal activity through bidirectional mechanisms.

The paper investigates how GABAARs on dopaminergic axons modulate nicotinic input integration, showing that GABAAR antagonism enhances nAChR-evoked activity.