Author

Christopher Foley

University of ArizonaORCID 0000-0002-9028-5841Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Aidan McMahon· 1 shared Arthur Y. Shaw· 1 shared Christopher HulmeiD· 1 shared Geidy E. SerranoiD· 1 shared Gerrit WilmsiD· 1 shared Jessica M. JuddiD· 1 shared Julie Turk· 1 shared Ragini Kushwaha· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Aidan McMahon

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1 paper

Recent·Most cited

Dyrk1a inhibition with the Novel Compound DYR533: A Cross-Disease Therapeutic Strategy Targeting Amyloidosis, Tau Pathogenesis, and Neuroinflammation

Samantha K. Bartholomew, Julie Turk, Wendy Winslow, Christopher Foley, Arthur Y. Shaw, Samantha Rokey, Savannah Tallino, Jessica M. Judd, Thomas G. Beach, Geidy E. Serrano, Gerrit Wilms, Ragini Kushwaha, Aidan McMahon, Sean Brendan Ginn, Walter B. Becker, , Ramón Velázquez

This study shows that inhibiting Dyrk1a with the compound DYR533 reduces tau pathology, neuroinflammation, and amyloidosis in mouse models of neurodegenerative diseases.

bioRxiv · Jan 23, 2026 · PreprintNeuroscience