Robert Park

The paper presents a pseudo-phased genome assembly of H. vastatrix isolate Hv178a, revealing a chromosomal trisomy of chr17 associated with increased virulence.

The paper tracks global spread and hybridization of wheat yellow rust pathogen, identifying nuclear reassortment and intercontinental movement as key drivers of evolution and disease impact.

This study presents a chromosome-level genome of Austropuccinia psidii, revealing its unique genomic features and infection dynamics across diverse hosts.

The paper presents a haplotype-resolved genome assembly of Puccinia hordei, revealing structural plasticity, copy number variation, and evolutionary dynamics in a barley fungal pathogen.

The paper uses parentally phased genomes of Theobroma cacao to identify NLR gene clusters at QTLs associated with resistance to vascular streak dieback.

The paper analyzes the genetic composition and history of the bread wheat D genome using a pangenome of its donor species, revealing contributions from discrete subpopulations of Aegilops tauschii.

This study identifies Rph7, a barley resistance gene against leaf rust, as a putative NAC transcription factor involved in basal defence activation.

The paper presents a chromosome-level genome assembly for the coffee rust pathogen Hemileia vastatrix, revealing gene duplication and potential symbiotic or contaminating fungal genomes.

The paper presents a chromosome-level phased genome and mitochondrial sequence for the dikaryotic rust fungus Austropuccinia psidii, revealing 18 chromosomes per nucleus and introducing a new tool for chromosome comparison.

This paper reports the assembly of a large, highly contiguous genome for Austropuccinia psidii, revealing its expansion through transposable elements and low GC content.

The paper develops and validates co-dominant PCR markers for the barley leaf rust resistance genes Rph20 and Rph24.

This study presents haplotype-phased genome assemblies of two dikaryotic Puccinia coronata f. sp. avenae isolates, revealing structural variation and heterozygous SNPs, and identifies co-expressed effector candidates associated with virulence differences.