Connor C. Leek

This study shows that the postnatal tendon enthesis remains hypoxic and requires Hif1a for cell survival, ECM organization, and structural integrity.

The paper develops patient-derived airway organoids from BAL fluid to model BPD injury and therapy responses in vitro.

This study uses scRNA-seq to analyze sex-based differences in neonatal lung injury recovery in mice, identifying transcriptional changes and cell-cell communication patterns influenced by biological sex.

This study shows that Fgf9 deletion in tendon disrupts enthesis mineralization by affecting chondrocyte hypertrophy and collagen type X.

The paper investigates sex-specific and cell-type specific transcriptional changes in neonatal hyperoxic lung injury using single-cell RNA sequencing in mice.

This study shows that deletion of Fgf9 in mice leads to enlarged deltoid tuberosities, suggesting a role for Fgf9 in muscle-bone interactions during development.