Elahe Ganji

Overexpression of enhanced yellow fluorescent protein fused with Channelrhodopsin-2 causes contractile dysfunction in skeletal muscle

This study shows that overexpressing ChR2-EYFP in skeletal muscle causes contractile dysfunction, while ChR2-only mice show normal function, highlighting the need for alternative optogenetic models.

Targeted Deletion of Fgf9 in Tendon Disrupts Mineralization of the Developing Enthesis

This study shows that Fgf9 deletion in tendon disrupts enthesis mineralization by affecting chondrocyte hypertrophy and collagen type X.

Deletion of Fibroblast growth factor 9 globally and in skeletal muscle results in enlarged tuberosities at sites of deltoid tendon attachments

This study shows that deletion of Fgf9 in mice leads to enlarged deltoid tuberosities, suggesting a role for Fgf9 in muscle-bone interactions during development.