Author

Michael D. Donnan

Northwestern UniversityORCID 0000-0002-9470-648XUnclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Amal Dadi· 1 shared Behram Radmanesh· 1 shared Benjamin FreedmaniD· 1 shared Byung Ha ChungiD· 1 shared Dan YiiD· 1 shared Dilip K. DebiD· 1 shared Esther LiuiD· 1 shared Jennie J. LiniD· 1 shared

Activity by Year

2020-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperGenomics

Amal Dadi

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2 papers

Recent·Most cited

VEGF-C overexpression in kidney progenitor cells is a model of renal lymphangiectasia

Michael D. Donnan, Dilip K. Deb, Valentin David, Susan E. Quaggin

This study creates a mouse model overexpressing VEGF-C in kidney progenitor cells, revealing increased lymphangiogenesis and a cystic kidney phenotype resembling human renal lymphangiectasia.

bioRxiv · May 3, 2023 · PreprintDevelopmental Biology

Profiling APOL1 Nephropathy Risk Variants in Genome-Edited Kidney Organoids with Single-Cell Transcriptomics

Esther Liu, Behram Radmanesh, Byung Ha Chung, Michael D. Donnan, Dan Yi, Amal Dadi, Kelly D. Smith, Jonathan Himmelfarb, Mingyao Li, Benjamin Freedman, Jennie J. Lin

The paper uses genome-edited human kidney organoids and single-cell transcriptomics to study APOL1 risk variants' effects on kidney disease mechanisms.

Kidney360 · Mar 1, 2020Peer-reviewedGenomics