Author

Ulrike Felgenhauer

Justus-Liebig-Universität GießenORCID 0000-0002-9478-5368Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Microbiology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Friedemann WeberiD· 2 shared David Poumo TchouassiiD· 1 shared Lars PelziD· 1 shared Markus HoffmanniD· 1 shared Matthew J. PickiniD· 1 shared Michael WinkleriD· 1 shared Najat BdeiriD· 1 shared Prerna AroraiD· 1 shared

Activity by Year

2021-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersMicrobiology

Friedemann WeberORCID

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2 papers

Recent·Most cited

Human cell line tropism, interferon system interactions and NSs mechanisms by the Ntepes and Gabek Forest phleboviruses

Matthew J. Pickin, Ulrike Felgenhauer, David Poumo Tchouassi, Sandra Junglen, Friedemann Weber

This paper investigates the cell line tropism, interferon system interactions, and NSs mechanisms of Ntepes and Gabek Forest phleboviruses, finding they can infect human cell lines and inhibit the interferon system similar to Rift Valley fever virus.

bioRxiv · Oct 17, 2025 · PreprintMicrobiology

Interferon induction and not replication interference mainly determines anti-influenza virus activity of defective interfering particles

Prerna Arora, Najat Bdeir, Sabine Gärtner, Stefanie Reiter, Lars Pelz, Ulrike Felgenhauer, Udo Reichl, Stephan Ludwig, Friedemann Weber, Markus Hoffmann, Michael Winkler, Stefan Pöhlmann

This study shows that defective interfering particles (DIPs) primarily exert antiviral activity through interferon (IFN) system activation rather than suppression of viral replication.

bioRxiv · Sep 20, 2021 · PreprintMolecular Biology