Author

Yutaka Maeda

Cincinnati Children's Hospital Medical CenterORCID 0000-0002-9537-3971Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Yutaka Maeda1 paper · Cincinnati Children's Hospital Medical Center

Biology Disciplines

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Bioengineering· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:deep learning· 1 Research Focus:differential expression· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

William D. StuartiD· 2 shared Anusha SridharaniD· 1 shared Assem G. ZiadyiD· 1 shared Bingxin LiuiD· 1 shared Donglu ShiiD· 1 shared Eric L. SnyderiD· 1 shared Hideo WatanabeiD· 1 shared I. M. Fink-Baldauf· 1 shared

Activity by Year

2024-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 27 mutual co-author links

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2 shared papersBioengineering

William D. StuartORCID

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Author profile →

2 papers

Recent·Most cited

PBAE-PEG based lipid nanoparticles for lung cell-specific gene delivery

Bingxin Liu, Yamato Sajiki, Nicole Littlefield, Yongxian Hu, William D. Stuart, Anusha Sridharan, Xuemei Cui, Matthew E. Siefert, Koichi Araki, Assem G. Ziady, Donglu Shi, Jeffery A Whitsett, Yutaka Maeda

This paper develops PBAE-PEG and PBAE lipid nanoparticles for targeted mRNA delivery to lung cells, showing differential efficacy in transfecting pulmonary endothelial and epithelial cells via different routes.

Molecular Therapy · Jan 25, 2025Peer-reviewedBioengineering

Patho-transcriptomic analysis of invasive mucinous adenocarcinoma of the lung (IMA): comparison with lung adenocarcinoma with signet ring cell features (SRCC)

William D. Stuart, Masaoki Ito, I. M. Fink-Baldauf, Takuya Fukazawa, Tomoki Yamatsuji, Tomoshi Tsuchiya, Hideo Watanabe, Morihito Okada, Eric L. Snyder, Mari A. Mino-Kenudson, Minzhe Guo, Yutaka Maeda

This study compares the patho-transcriptomic profiles of invasive mucinous adenocarcinoma (IMA) and adenocarcinoma with signet ring cell features (SRCC), identifying distinct and shared gene expression patterns and potential therapeutic targets.

bioRxiv · Jun 13, 2024 · PreprintCancer Biology