Uta Mamrak

This study identifies cerebral microthrombi as focal sites of Cav-1-dependent BBB dysfunction after TBI, linking microvascular thrombosis to neuronal damage through transcytosis.

This paper develops a real-time FRET-based system to monitor nanocarrier behavior and drug release at post-stroke microthrombi in vivo.

This study develops an optimized surgical method to reduce infarct volume variability in C57BL/6 mice by preventing PCA area infarcts and improving reproducibility in the fMCAo model.

The paper shows pericytes constrict during ischemia, remain constricted post-reperfusion, and contribute to no-reflow, with some surviving and upregulating transcripts, suggesting they are therapeutic targets.

This study shows that glycolic acid protects neurons from ischemic damage by reducing calcium-mediated excitotoxicity during reperfusion in stroke models.