Author

Isabella S. Del Priore

Memorial Sloan Kettering Cancer CenterORCID 0000-0002-9665-1101Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genetics· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Research Focus:differential expression· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Assay:multiome· 1

Frequent Co-Authors

Ahmed M. ElhossinyiD· 1 shared Andrea C. ChaikovskyiD· 1 shared Andrew R Moorman· 1 shared Aveline A. FillioliD· 1 shared Cassandra BurdziakiD· 1 shared Catherine H. FreudenreichiD· 1 shared Catherine Snopkowski· 1 shared Dana Pe’eriD· 1 shared

Activity by Year

2023-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperCancer Biology

Ahmed M. ElhossinyORCID

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2 papers

Recent·Most cited

Oncogenic and tumor-suppressive forces converge on a progenitor niche at the benign-to-malignant transition

Jose Reyes, Isabella S. Del Priore, Andrea C. Chaikovsky, Nikhita Pasnuri, Ahmed M. Elhossiny, Jin Park, Philipp Weiler, Tobias Krause, Andrew R Moorman, Catherine Snopkowski, Meril T. Takizawa, Cassandra Burdziak, Nalin Ratnayeke, Ignas Masilionis, Yu-Jui Ho, , Scott W. Lowe

In mouse pancreatic cancer models, p53, CDKN2A and SMAD4 programs converge on a rare Kras-mutant progenitor-like state whose niche KRAS inhibition collapses and p53 loss expands.

Cell · Apr 15, 2026Peer-reviewedCancer Biology✦ In-depth digest

Structure-forming CAG/CTG repeats interfere with gap repair to cause repeat expansions and chromosome breaks

Erica J. Polleys, Isabella S. Del Priore, Marjorie de la Rosa Mejia, James E. Haber, Catherine H. Freudenreich

The paper shows that CAG/CTG repeats in ssDNA gaps influence repair kinetics, leading to repeat expansions or contractions and chromosome breaks.

Nature Communications · Apr 29, 2023Peer-reviewedGenetics