Claudio V. Mello

The paper shows that GPR108 absence in birds explains lower AAV transduction efficacy and demonstrates that human GPR108 expression and capsid modifications enhance transduction in zebra finch cells.

The paper identifies a direct cortical projection from the primary motor cortex to the nucleus ambiguus in two bat species, suggesting they have the anatomical substrate for vocal learning.

This study shows that a learned motor behavior can recover after chronic disruption of inhibitory circuits, with neuronal activity permanently altered but song recovery possible without relying on learning circuits.

The paper identifies Kv3.1 as a key channel enabling ultranarrow spikes in songbird RAPNs, analogous to Betz cells in primates, supporting precise motor control.

The paper evaluates new vertebrate genome assemblies from the VGP against previous references, finding significant improvements in completeness and accuracy.

This study identifies resurgent Na+ currents in zebra finch projection neurons that enable ultrafast spiking for fine motor control.

This study shows that the loss of ephrin-B3 function in birds shapes spinal flight circuitry by enabling synchronous wing flapping through altered midline signaling.

The paper summarizes methods and lessons from assembling 16 vertebrate genomes, highlights the importance of long-read sequencing, and introduces the Vertebrate Genomes Project to generate complete reference genomes for all vertebrate species.