Author

Matthew Alsaloum

Brigham and Women's HospitalORCID 0000-0002-9832-0138Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1 Organism:mouse· 1

Frequent Co-Authors

Fadia B. Dib-HajjiD· 1 shared Grant P. Higerd‐RusliiD· 1 shared Mohammad‐Reza GhovanlooiD· 1 shared Peng ZhaoiD· 1 shared Philip R. EffraimiD· 1 shared Shujun LiuiD· 1 shared Sidharth TyagiiD· 1 shared Stephen G. WaxmaniD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperNeuroscience

Fadia B. Dib-HajjORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Targeted ubiquitination of NaV1.8 reduces sensory neuronal excitability

Sidharth Tyagi, Mohammad‐Reza Ghovanloo, Matthew Alsaloum, Philip R. Effraim, Grant P. Higerd‐Rusli, Fadia B. Dib-Hajj, Peng Zhao, Shujun Liu, Stephen G. Waxman, Sulayman D. Dib‐Hajj

This study develops a therapeutic protein, UbiquiNaV, that reduces NaV1.8 channel expression in sensory neurons, alleviating pain in preclinical models.

bioRxiv · Feb 4, 2025 · PreprintNeuroscience