Author

Sulayman D. Dib‐Hajj

Yale UniversityORCID 0000-0002-4137-1655Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:drug response / drug discovery· 1 Organism:human· 1

Frequent Co-Authors

Stephen G. WaxmaniD· 2 shared Arthur Silveira PrudenteiD· 1 shared Beatriz AdjafreiD· 1 shared Conceição Elidianne Anibal Silva· 1 shared Danilo Roman‐CamposiD· 1 shared Fadia B. Dib-HajjiD· 1 shared Fernando Q. CunhaiD· 1 shared Francisco Isaac Fernandes GomesiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 31 mutual co-author links

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2 shared papersNeuroscience

Stephen G. WaxmanORCID

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2 papers

Recent·Most cited

Nav1.5 expressed in TrkB+ sensory neurons mediates paclitaxel-induced mechanical pain hypersensitivity

Francisco Isaac Fernandes Gomes, Beatriz Adjafre, Sang Hoon Lee, Arthur Silveira Prudente, Gabriel V. Lucena-Silva, Conceição Elidianne Anibal Silva, Luiz Kanada, Danilo Roman‐Campos, Jose C Alves-Filho, Fernando Q. Cunha, Stephen G. Waxman, Sulayman D. Dib‐Hajj, Temugin Berta, Thiago M Cunha

This study identifies Nav1.5 in TrkB+ sensory neurons as a driver of paclitaxel-induced mechanical pain hypersensitivity in mice, with potential translational relevance to humans.

bioRxiv · Mar 23, 2026 · PreprintNeuroscience

Targeted ubiquitination of NaV1.8 reduces sensory neuronal excitability

Sidharth Tyagi, Mohammad‐Reza Ghovanloo, Matthew Alsaloum, Philip R. Effraim, Grant P. Higerd‐Rusli, Fadia B. Dib-Hajj, Peng Zhao, Shujun Liu, Stephen G. Waxman, Sulayman D. Dib‐Hajj

This study develops a therapeutic protein, UbiquiNaV, that reduces NaV1.8 channel expression in sensory neurons, alleviating pain in preclinical models.

bioRxiv · Feb 4, 2025 · PreprintNeuroscience