Kunimasa Suzuki

This study investigates GlyR dysfunction in T2D and high glucose conditions, finding reduced GlyR currents and gene expression, but no splicing changes, and links GlyR activity to gene regulatory networks.

This study identifies TMEM55A as a regulator of α-cell exocytosis and glucagon secretion through PI5P signaling and F-actin remodeling in response to oxidative stress.

This study links transcriptomic heterogeneity in pancreatic islet cells to their physiological function using a combined electrophysiological and single-cell RNA sequencing approach, identifying genes and pathways associated with diabetes-related dysfunction.