Author

Josh Ivie

Seattle Children's Research InstituteORCID 0000-0003-0116-1781Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Genetics· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Alan H. DiercksiD· 1 shared Alexis KaushanskyiD· 1 shared Alissa C. RothchildiD· 1 shared Alwyn M. V. EckeriD· 1 shared Benjamin Henry GerniD· 1 shared Catherine M. SteiniD· 1 shared Charlie J. PyleiD· 1 shared Clare M. SmithiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperImmunology

Alan H. DiercksORCID

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintJun 4, 2026

Modulation of Ferroptosis During Early Mycobacterium tuberculosis Infection Contributes to Beijing Lineage Strain SA161 Virulence

Josh Ivie, Sylvia M. Stull, Ethan Bustad…·Shuyi Ma

This study shows that ferroptosis, a form of cell death, is enhanced by a virulent Mtb strain, SA161, and inhibiting it reduces bacterial spread in mice.

DOI 10.64898/2026.05.29.728786

mousescRNA-seq

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GeneticsPLoS BiologyPeer-reviewedSep 9, 2025

Cathepsin Z is a conserved susceptibility factor underlying tuberculosis severity

Rachel K. Meade, Oyindamola O. Adefisayo, Marco Túlio Pardini Gontijo…·Clare M. Smith

This study identifies cathepsin Z as a susceptibility factor for tuberculosis severity in both mice and humans, showing its role in disease outcomes through genetic and cellular analyses.

3 citations

mousehuman

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