Author

Giorgia Farinacci

Sapienza University of RomeORCID 0000-0003-0122-0155Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Organism:mouse· 3 Approach:experimental assay· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Assay:imaging· 1 Approach:statistical model· 1

Frequent Co-Authors

Alessandro CorsiiD· 3 shared Biagio PalmisanoiD· 3 shared Chiara Tavanti· 3 shared Mara RiminucciiD· 3 shared Marta SerafiniiD· 2 shared Natasha M. Appelman‐DijkstraiD· 2 shared Samantha DonsanteiD· 2 shared Alessia CiarrocchiiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 42 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

3 shared papersPathology

Alessandro CorsiORCID

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

PathologyCommunications BiologyPeer-reviewedApr 22, 2026

Exploring the mechanism and pattern of bone formation during RANKL inhibition in a mouse model of fibrous dysplasia

Giorgia Farinacci, Ilenia Coletta, Biagio Palmisano…·Mara Riminucci

This study investigates how RANKL inhibition affects bone formation in a mouse model of fibrous dysplasia, finding that bone formation is restricted to specific surfaces rather than occurring diffusely.

1 citation

mousestatistical model

𝕏 PostBluesky

PathologyJournal of Bone and Mineral ResearchPeer-reviewedFeb 22, 2026

Effects of anti-RANKL, Zoledronate or combination therapy in a mouse model of Fibrous Dysplasia: a preclinical study

Biagio Palmisano, Chiara Tavanti, Giorgia Farinacci…·Mara Riminucci

This study evaluates the combination of zoledronate and anti-RANKL therapy in a mouse model of fibrous dysplasia, showing delayed disease rebound and improved bone density.

3 citationstop 1%

mouseexperimental assay

𝕏 PostBluesky

PathologyJournal of Bone and Mineral ResearchPeer-reviewedMay 3, 2025

Bone pain in Fibrous dysplasia does not rely on aberrant sensory nerve sprouting or neuroma formation

Biagio Palmisano, Chiara Tavanti, Giorgia Farinacci…·Mara Riminucci

This study shows that bone pain in Fibrous dysplasia is intrinsic and not caused by abnormal sensory nerve sprouting or neuroma formation.

8 citationstop 3%

mousehuman

𝕏 PostBluesky