Terri Pietka

The paper shows immune cells regulate circulating adipocyte extracellular vesicle levels by clearing EVs, with reduced clearance in obesity leading to increased circulating adipoEVs.

This study shows that overexpressing NAMPT in adipocytes protects against metabolic dysfunction in male obese mice but not females, via improved interorgan communication and extracellular vesicle effects.

This study identifies nSMase2-generated exosomes as a mechanism for adipocyte LPL secretion and transfer to endothelial cells via GPIHBP1.

This study identifies Site-1 Protease (S1P) as a negative regulator of muscle mass and mitochondrial metabolism by controlling the TGF-β target gene MSS51.

This study shows that Mogat1 is important for adipocyte differentiation in vitro but not for adiposity or metabolic outcomes in mice on a high-fat diet.