Author

Agnieszka Witalisz‐Siepracka

Karl Landsteiner University of Health SciencesORCID 0000-0003-0237-0745Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell signaling & trafficking· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:wet-lab / biochemical assay· 1

Frequent Co-Authors

Alexandra DrakakiiD· 1 shared Dagmar StoiberiD· 1 shared Emiel P. C. van der VorstiD· 1 shared Josje M.A. HuismaniD· 1 shared Marten A. HoeksemaiD· 1 shared Mees Botman· 1 shared Ricky Siebeler· 1 shared Sanne Lidewij MaasiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 28 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

Alexandra DrakakiORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Uncovering an unconventional JAK1/2-STAT3 branch in macrophage IFNγ signaling

Mees Botman, Josje M.A. Huisman, Alexandra Drakaki, Sanne Lidewij Maas, Agnieszka Witalisz‐Siepracka, Dagmar Stoiber, Emiel P. C. van der Vorst, Ricky Siebeler, Marten A. Hoeksema

The paper identifies an unconventional JAK1/2-STAT3 signaling pathway in macrophage IFNγ responses, showing STAT3 activation and heterodimerization with STAT1, but limited gene regulatory contribution.

bioRxiv · Aug 13, 2025 · PreprintImmunology