Author

Wei Cheng

University of MichiganORCID 0000-0003-0332-1566Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Wei Cheng3 papers · Shanghai Medical College of Fudan University

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Assay:metagenomics· 2 Assay:proteomics· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

Alexander R. MeyeriD· 2 shared James L. Mueller· 2 shared Julie ZikhermaniD· 2 shared Wei-Yun WholeyiD· 2 shared Alexander J. RitteriD· 1 shared Bryce ChackerianiD· 1 shared Erika Kay-TsumagariiD· 1 shared François X. P. BourassaiD· 1 shared

Activity by Year

2024-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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2 shared papersImmunology

Alexander R. MeyerORCID

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2 papers

Recent·Most cited

ImmunologybioRxivPreprintApr 24, 2026

Virus-like antigen display delivers a stand-alone danger signal through the BCR that circumvents tolerance

Julianne B. Riggs, Alexander J. Ritter, François X. P. Bourassa…·Julie Zikherman

The paper shows that virus-like particle-displayed antigens bypass BCR tolerance by triggering distinct signaling and transcriptional programs compared to soluble antigens.

DOI 10.64898/2026.04.23.720482

proteomicsmetagenomics

𝕏 PostBluesky

ImmunologyThe Journal of ImmunologyPeer-reviewedAug 30, 2024

An integrated signaling threshold initiates IgG response towards virus-like immunogens

Wei-Yun Wholey, Alexander R. Meyer, Sekou-Tidiane Yoda…·Wei Cheng

The paper identifies that a combination of surface antigen density and internal nucleic acids is sufficient to initiate strong antiviral IgG responses without T cell help or Toll-like receptor signaling.

9 citations

mouseproteomics

𝕏 PostBluesky