Author

Tom Winkler

Tel Aviv UniversityORCID 0000-0003-0333-9328Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Tom Winkler1 paper · University of Cologne

Biology Disciplines

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Cancer Biology· 3

Signature Assays, Organisms & Methods

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Organism:human· 3 Approach:statistical model· 3 Assay:bulk RNA-seq· 2 Research Focus:drug response / drug discovery· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:chromatin & epigenetic regulation· 1 Approach:experimental assay· 1 Research Focus:variant calling· 1

Frequent Co-Authors

Uri Ben‐DavidiD· 3 shared Gil LeoriD· 2 shared Adi V Tarrab· 1 shared Eli ReuveniiD· 1 shared Eran SdeoriD· 1 shared Geert J.P.L. KopsiD· 1 shared Guy Wolf· 1 shared Guy Wolf-Dankovich· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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3 shared papersCancer Biology

Uri Ben‐DavidORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 30, 2026

Mapping chromothripsis across human cancer cell lines reveals genome evolution and therapeutic vulnerabilities

Gil Leor, Tal Ben-Yishay, Adi V Tarrab…·Uri Ben‐David

This study maps chromothripsis in 1,646 human cancer cell lines, showing its conservation with tumors, higher prevalence, and therapeutic vulnerabilities to HDAC inhibitors.

DOI 10.64898/2026.10.30.763015

humanwhole-genome sequencing

𝕏 PostBluesky

Cancer BiologybioRxivPreprintDec 2, 2025

Chromosome-arm 17p Loss Renders Breast Cancer Cells Vulnerable to AURKB Inhibition

Tom Winkler, Eran Sdeor, Ron Saad…·Uri Ben‐David

This study identifies Del17p as a biomarker for AURKB inhibition in breast cancer, showing increased sensitivity to AURKB inhibitors in Del17p tumors.

DOI 10.64898/2025.12.01.691489

humanbulk RNA-seq

𝕏 PostBluesky

Cancer BiologybioRxivPreprintJul 4, 2025

p53 is a selective barrier to chromosome loss in cancer

Joana Rita Fernandes Marques, Marco António Dias Louro, Michaël Schubert…·Geert J.P.L. Kops

This study shows p53 inactivation is not universally linked to aneuploidy but is associated with increased chromosome loss in cancer.

DOI 10.1101/2025.07.04.663162

humanbulk RNA-seq

𝕏 PostBluesky