Khadija Wahni

This study shows disease-linked mutations in hnRNPA2B1 accelerate condensate maturation and promote amyloid aggregation by altering phase separation and fibril formation.

This study attempts to reconstitute the transient Prdx2:STAT3 redox-relay complex in vitro but finds it challenging due to its redox-dependent nature and susceptibility to hyperoxidation.

The paper shows that eukaryotic Prx1/AhpC-type peroxiredoxins form heterooligomers, which modulate their structure and stability, challenging the previous belief in homooligomerization.