Author

Eva Santamaría

Centro de Investigación Biomédica en Red de Enfermedades Hepáticas y DigestivasORCID 0000-0003-0629-6175Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Genomics· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Research Focus:gene regulatory network inference· 2 Organism:human· 2 Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1

Frequent Co-Authors

Bruno SangroiD· 2 shared Josepmaria ArgemíiD· 2 shared Pablo SarobeiD· 2 shared Puri FortesiD· 2 shared Agavni MesropianiD· 1 shared Albert Gris‐OliveriD· 1 shared Alexander FinnemoreiD· 1 shared Alexis I. CocozakiiD· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersCancer Biology

Bruno SangroORCID

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3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 16, 2026

ATF4 programs proline-dependent immune evasion in β-Catenin-driven hepatocellular carcinoma

Stefany Infante, Eva Santa María, Alexander Finnemore…·Josepmaria Argemí

This study identifies ATF4 as a key driver of immune evasion in β-catenin-driven hepatocellular carcinoma by programming a proline-dependent metabolic axis that suppresses anti-tumor immunity.

DOI 10.64898/2026.05.12.724215

humanmouse

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Pharmacology & ToxicologyCell Reports MedicinePeer-reviewedMar 31, 2026

Histone methyl-transferase G9a inhibition boosts the efficacy of immune checkpoint inhibitors in experimental hepatocellular carcinoma

Elena Adán-Villaescusa, Borja Castelló-Uribe, Iker Uriarte…·Maite G Fernandez-Barrena

This study shows that inhibiting G9a enhances the effectiveness of immune checkpoint inhibitors in hepatocellular carcinoma by improving tumor immunogenicity and T cell infiltration.

3 citationstop 2%

mousebulk RNA-seq

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GenomicsbioRxivPreprintMar 16, 2026

Oncogenes and tumor suppressor genes are enriched in stop-loss mutations generating protein extensions

Lilian Marie Boll, Joan Antoni Martorell, Nafiseh Khelghati…·M. Mar Albà

The paper identifies stop-loss mutations in cancer genomes, showing enrichment in cancer-related genes and potential immunological consequences.

DOI 10.64898/2026.03.12.711331

humanwhole-genome sequencing

𝕏 PostBluesky