Bernhard Bettler

This study shows that APP17, a peptide derived from APP, does not regulate GABAB receptor activity in heterologous cells or neurons, suggesting sAPP acts through other receptors.

The paper identifies a GABA/TNF-related cytokine-mediated communication pathway between interneurons and oligodendrocyte precursor cells that affects myelination and cognitive behavior.

This study shows that GABAB receptor/HCN channel complexes in VTA dopamine neurons limit synaptic inhibition and reduce anxiety-like behavior by modulating K+ currents.

The paper investigates how GABAB receptor auxiliary subunits modulate Cav2.3-mediated neurotransmitter release in medial habenula terminals, revealing their role in synaptic strength regulation.