Author

Michael I. Krauss

Leibniz-Forschungsinstitut für Molekulare PharmakologieORCID 0000-0003-1010-9723Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Approach:genetic perturbation / CRISPR· 1 Organism:human· 1

Frequent Co-Authors

Christopher SchmiediD· 1 shared Florian HeydiD· 1 shared Giulia RussoiD· 1 shared Helge EwersiD· 1 shared Martin LehmanniD· 1 shared Nadja HümpferiD· 1 shared Nina Jaensch· 1 shared Steffen D. ResteliD· 1 shared

Activity by Year

2026-2026 · peak 1/yr

Co-Author Collaboration Network

9 top collaborators · 36 mutual co-author links

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1 shared paperCell Biology

Christopher SchmiedORCID

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1 paper

Recent·Most cited

Septin-associated PIPKIγ splice variants drive centralspindlin association with the midbody via PI(4,5)P2

Giulia Russo, Nadja Hümpfer, Nina Jaensch, Steffen D. Restel, Christopher Schmied, Florian Heyd, Martin Lehmann, Helge Ewers, Volker Haucke, Michael I. Krauss

This study identifies septin-associated PIPKIγ splice variants as essential for midbody organization by generating PI(4,5)P2 required for centralspindlin recruitment and septin-microtubule association.

Nature Communications · Feb 7, 2026Peer-reviewedCell Biology