Carlos Fernando Valenzuela

This study examines the effects of third-trimester-equivalent ethanol exposure on neuronal and VGLUT2+ cell density in the dorsal subiculum of male and female mice, finding sex-specific changes in neuronal density and excitability.

The paper identifies epileptiform discharges as a critical feature in a preclinical model of FASD, showing their relationship to high-frequency oscillations in the retrosplenial cortex.

This study shows that third-trimester-equivalent alcohol exposure in neonatal mice leads to sex-dependent behavioral changes and increased mechanical allodynia in adulthood.

This study shows ethanol increases GABAA receptor-mediated spontaneous postsynaptic currents in immature mouse cerebellar Golgi cells, enhancing inhibitory input to granule cells.

This study shows prenatal alcohol exposure enhances morphine-induced pathological pain through NLRP3-mediated proinflammatory immune responses in nerve-injured mice.

The paper uses in vivo electrophysiology and dimensionality reduction to show alcohol exposure reduces firing rates in thalamic neurons but increases hippocampal excitability in mice.

The paper investigates how ethanol exposure disrupts neuronal networks involved in fear memory in mice, finding hyperconnectivity in the limbic system and altered Fos+ neuron activity.

This study examines how different patterns of maternal alcohol consumption during pregnancy affect offspring neurobehavioral outcomes in mice.

This study examines the effects of neonatal ethanol exposure on GABAergic neurotransmission in the retrosplenial cortex of adolescent mice, finding altered parvalbumin interneuron function.