Author

Divya Sahu

University of Alabama at BirminghamORCID 0000-0003-1246-8412Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Approach:statistical model· 1

Frequent Co-Authors

Anindya DuttaiD· 2 shared Ajay ChatrathiD· 1 shared Chase A. WeidmanniD· 1 shared Fatemeh EsmaeiliiD· 1 shared Isaac K. NardiiD· 1 shared Kevin M. WeeksiD· 1 shared Kumarjeet Bannerjee· 1 shared Mujawar Aaiyas· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 25 mutual co-author links

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2 shared papersCancer Biology

Anindya DuttaORCID

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintJun 24, 2026

tRF-3021a Supports Invasiveness, Apoptosis Resistance, and Global Protein Synthesis in Cancer Cell Lines

Fatemeh Esmaeili, Kumarjeet Bannerjee, Ajay Chatrath…·Anindya Dutta

The paper identifies tRF-3021a as a cancer-promoting tRNA-derived fragment that supports invasiveness, apoptosis resistance, and global protein synthesis in cancer cell lines.

DOI 10.64898/2026.01.26.701835

humanbulk RNA-seq

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Molecular BiologyJournal of Biological ChemistryPeer-reviewedMay 2, 2025

A 36-base hairpin within lncRNA DRAIC, which is modulated by alternative splicing, interacts with the IKKα coiled-coil domain and inhibits NF-κB and tumor cell phenotypes

Xiaoxiao Hao, Yuechuan Chen, Divya Sahu…·Anindya Dutta

The paper identifies a 36-base hairpin in lncRNA DRAIC that interacts with IKKα's coiled-coil domain, inhibits NF-κB activity, and suppresses tumor phenotypes through alternative splicing regulation.

3 citations

humanbulk RNA-seq

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