Author

Masaki Imanishi

The University of Texas MD Anderson Cancer CenterORCID 0000-0003-1305-5943Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Molecular Biology· 1 Pharmacology & Toxicology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Anita M DeswaliD· 1 shared Criag Morrell· 1 shared Eduardo N. ChiniiD· 1 shared Efstratios KoutroumpakisiD· 1 shared Erin H. SeeleyiD· 1 shared Gilbert F. MejiaiD· 1 shared Guangyu WangiD· 1 shared Haodong XuiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 36 mutual co-author links

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1 shared paperMolecular Biology

Anita M DeswalORCID

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2 papers

Recent·Most cited

Pharmacology & ToxicologyHORMONESPeer-reviewedJul 8, 2026

Highly selective SGLT2 inhibitors suppress glucose uptake in alpha-TC1 cells, while glucagon secretion is not affected.

Licht Miyamoto, Suguru Nakayama, Honoka Endoh…·Koichiro Tsuchiya

This study investigates the effects of SGLT2 inhibitors on glucose uptake and glucagon secretion in alpha-TC1 cells, finding that SGLT2 mediates basal glucose uptake but not glucagon secretion.

1 citation

experimental assayhost-pathogen & immune response

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Molecular BiologybioRxivPreprintJun 19, 2025

Downregulation of LATS1/2 Drives Endothelial Senescence-Associated Stemness (SAS) and Atherothrombotic Lesion Formation

Sivareddy Kotla, Jonghae Lee, Kyung Ae Ko…·Jun‐ichi Abe

This study shows that loss of LATS1/2 in endothelial cells promotes a senescence-associated stemness (SAS) state, leading to atherothrombotic plaque formation through metabolic and mitochondrial dysregulation.

1 citation

mousehuman

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