Author

Kun Gao

University of California, Los AngelesORCID 0000-0003-1400-286XUnclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Kun Gao1 paper · Southeast University

Biology Disciplines

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Neuroscience· 3

Signature Assays, Organisms & Methods

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Organism:mouse· 3 Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Research Focus:gene regulatory network inference· 2 Assay:ATAC-seq· 1 Approach:benchmark study· 1 Approach:statistical model· 1

Frequent Co-Authors

Daniel H. GeschwindiD· 3 shared Clifford J. WoolfiD· 2 shared Riki KawaguchiiD· 2 shared Yuyan ChengiD· 2 shared Zhigang HeiD· 2 shared Aboozar MonavarfeshaniiD· 1 shared Alexander M. Bernstein· 1 shared Alice ZhangiD· 1 shared

Activity by Year

2017-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 39 mutual co-author links

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3 shared papersNeuroscience

Daniel H. GeschwindORCID

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3 papers

Recent·Most cited

NeuroscienceNature CommunicationsPeer-reviewedJul 29, 2022

Transcription factor network analysis identifies REST/NRSF as an intrinsic regulator of CNS regeneration

Yuyan Cheng, Yuqin Yin, Alice Zhang…·Daniel H. Geschwind

The paper identifies REST/NRSF as a suppressor of CNS axon regeneration using systems biology approaches and validates its role through genetic manipulations.

69 citationstop 2%

mousebulk RNA-seq

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NeuroscienceNeuronPeer-reviewedJun 28, 2022

Core Transcription Programs Controlling Injury-Induced Neurodegeneration of Retinal Ganglion Cells

Feng Tian, Yuyan Cheng, Songlin Zhou…·Zhigang He

The paper identifies four ATF/CEBP transcription factors that drive injury-induced neurodegeneration of retinal ganglion cells and shows their role in glaucoma.

128 citationstop 1%

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NeuroscienceeLifePeer-reviewedDec 19, 2017

Inducible and reversible phenotypes in a novel mouse model of Friedreich’s Ataxia

Vijayendran Chandran, Kun Gao, Vivek Swarup…·Daniel H. Geschwind

This study develops an inducible mouse model of Friedreich’s Ataxia to investigate reversible phenotypes by modulating frataxin levels and observing functional recovery.

99 citationstop 4%

mouseexperimental assay

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