Author

Paul Alayrac

InsermORCID 0000-0003-1686-7496Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 3 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1

Frequent Co-Authors

Stéphane M. CamusiD· 3 shared Camille A. KnospiD· 2 shared Clément CochainiD· 2 shared Jean‐Sébastien SilvestreiD· 2 shared Marie PiolletiD· 2 shared Adrien Tin-Kim-Wang· 1 shared Alain TedguiiD· 1 shared Alexander Michael Leipold· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersImmunology

Stéphane M. CamusORCID

Mutual co-authors in cluster

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3 papers

Recent·Most cited

Cell BiologybioRxivPreprintOct 2, 2026

Cell-specific isolation and functional analysis of primary cardiomyocyte-derived large extracellular vesicles from infarcted mouse hearts

Lélia Borowski, Alina Ramazanova, Paul Alayrac…·Stéphane M. Camus

This paper develops a method to isolate cell-type-specific extracellular vesicles from infarcted mouse hearts and demonstrates their functional properties.

DOI 10.64898/2026.10.01.755891

mouseproteomics

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ImmunologybioRxivPreprintAug 21, 2026

TREM2 drives accumulation of pro-scarring monocyte-derived macrophages in the infarcted myocardium

Giuseppe Rizzo, Marie Piollet, Tobias Krammer…·Clément Cochain

This study shows TREM2 drives pro-scarring macrophage accumulation in infarcted myocardium, leading to fibrosis and reduced cardiac repair.

DOI 10.64898/2026.08.14.744182

mousespatial transcriptomics

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ImmunologybioRxivPreprintJan 21, 2026

Mucosal-Associated Invariant T Cells Promote Atherosclerosis Through Monocyte-Driven Inflammation

Tobias Radecke, Thomas Nipoti, Louise Z Wang…·Soraya Taleb

This study identifies MAIT cells as contributors to atherosclerosis through monocyte-driven inflammation, showing their enrichment in plaques and protective effects when depleted.

DOI 10.64898/2026.01.19.700259

humanmouse

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